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Fri Dec 27, 2013 11:24 am  |
| i bought that eyezone microcurrent little machine for $34 but I HATE getting shocked. I like this little device by truthinaging. Its ultrasonic and there are 3 light modes; blue for acne, green for hyperpigmentation and green for skin plumping and wrinkles.It doesnt hurt at all and Marta from truth in aging has a 3 minute youtube on instructions,only takes 4 minutes for the whole face and does provide lift.I actually was able to buy it on ebay for only 59. |
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Fri Dec 27, 2013 11:55 am |
Lucy if you are still getting shocked by the EyeZone device, turn it down to a lower strength. You should not feel the current.
Also, of course you keep the area being treated moist with water or whatever to facilitate the current, right? I think its a neat little device and thanks for finding it! Love it! For $14 whats not to like? 
LED has been implicated in free radical generation so Im not interested in that at all. |
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Fri Dec 27, 2013 12:31 pm |
| fawnie wrote: |
Lucy if you are still getting shocked by the EyeZone device, turn it down to a lower strength. You should not feel the current.
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I 2nd this...the setting should only be at 1 or 2. |
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No longer answering PM's due to numerous weird messages. |
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Fri Dec 27, 2013 2:32 pm |
 free radical generation????OMG!thats the first I hear of this. would microcurrent do the same thing? |
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Fri Dec 27, 2013 3:20 pm |
| lucyluc wrote: |
| free radical generation????OMG!thats the first I hear of this. would microcurrent do the same thing? |
I think microcurrent has some good research behind it, but someone else can link to that. This is some research about infrared light (like LED devices have):
Effects of infrared radiation and heat on human skin aging in vivo.
Cho S, Shin MH, Kim YK, Seo JE, Lee YM, Park CH, Chung JH.
Source
Department of Dermatology, Seoul National University College of Medicine, Seoul, Korea.
Abstract
Sunlight damages human skin, resulting in a wrinkled appearance. Since natural sunlight is polychromatic, its ultimate effects on the human skin are the result of not only the action of each wavelength separately, but also interactions among the many wavelengths, including UV, visible light, and infrared (IR). In direct sunlight, the temperature of human skin rises to about 40 degrees C following the conversion of absorbed IR into heat. So far, our knowledge of the effects of IR radiation or heat on skin aging is limited. Recent work demonstrates that IR and heat exposure each induces cutaneous angiogenesis and inflammatory cellular infiltration, disrupts the dermal extracellular matrix by inducing matrix metalloproteinases, and alters dermal structural proteins, thereby adding to premature skin aging. This review provides a summary of current research on the effects of IR radiation and heat on aging in human skin in vivo. Journal of Investigative Dermatology Symposium Proceedings (2009) 14, 15-19; doi:10.1038/jidsymp.2009.7.
High-dose long wave visible light induces perinuclear vacuolization in vivo but does not result in early photoageing and apoptosis.
Tjioe M, Smits T, Blokx WA, van de Kerkhof PC, Gerritsen MJ.
Source
Department of Dermatology, University Medical Centre Nijmegen, The Netherlands. M.Tjioe@derma.umcn.nl
Abstract
With the advancing widespread use of photodynamic therapy, questions have arisen about the necessity to protect the adjacent healthy skin from high-dose long-wave light. The aim of the present study was to investigate the effects of high dose visible light on the skin of healthy volunteers with focus on apoptosis, DNA damage, inflammation, melanogenesis and induction of matrix metalloproteinases (MMP). Fourteen healthy volunteers were included and irradiated daily on their buttocks with 1300 kJ/m2 long wave visible light (560-780 nm) on five consecutive days with a cumulative dose of 6500 kJ/m2. In each volunteer six biopsies were taken before and 24 h after irradiation on days 1, 2, 3 and 5 and on day 8 and 12. Frozen and paraffin sections were investigated by measuring parameters for photodamage (apoptosis, p53, phosphorylated c-Jun), skin ageing (phosphorylated c-Jun, MMP-1, elastin content) melanogenesis (Melan A). Although no sunburn cells were seen, a significant increase in perinuclear vacuolization was noted (P < 0.0003) from day 5 till 7 days after the last irradiation. There was no expression of phosphorylated c-Jun, whereas the expression of p53, Melan A, MMP-1 and elastin content did not change. High-dose visible light induces a significant increase in perinuclear vacuolization, but does not result in apoptosis, photodamage or early induction of skin ageing.
The pathogenesis of photoaging: the role of neutrophils and neutrophil-derived enzymes.
Rijken F, Bruijnzeel PL.
Source
Department of Dermatology, University Medical Center Utrecht, Heidelberglaan, Utrecht, The Netherlands. F.rijken@umcutrecht.nl
Abstract
The hallmark of photoaged skin is solar elastosis, which is probably an end product of elastic fiber degradation. Exposure of human skin to a certain threshold of UV, infrared radiation (IR), and heat leads to an influx of neutrophils. These neutrophils are packed with potent proteolytic enzymes capable of degrading collagen and, particularly, elastic fibers. Neutrophil-derived proteolytic enzymes are held responsible for the extracellular matrix (ECM) damage observed in several non-dermatological conditions. Furthermore, neutrophil elastase, a major product of neutrophils, is strongly associated with solar elastosis in mice. Taken together with our data that show in vivo proteolytic activity of neutrophil-derived elastase and matrix metalloproteinases (MMPs) in UV-exposed skin, we have hypothesized earlier that neutrophils are major contributors to the photoaging process. Although several groups have shown that MMPs are also induced in skin exposed to relatively low doses of UV, IR, and heat, clinical data indicate that high(er) doses of UV, IR, and heat are necessary to induce photoaging or photoaging-like pathology in the skin. Therefore, we propose that MMPs generated by suberythemogenic doses of UV and low doses of IR/heat are involved in cellular processes other than ECM degradation.Journal of Investigative Dermatology Symposium Proceedings (2009) 14, 67-72; doi:10.1038/jidsymp.2009.15. |
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